This assumption essentially infers that gene interactions and epistatic effects may be captured by taking into account a number of selected variants that may be interacting with each other under an additive hypothesis. phenotypic variation of ?3.5) and may indicate a pathogenic role of T-cells and T-cells differentiation in the disease, suggesting an immune mechanism … Continue reading This assumption essentially infers that gene interactions and epistatic effects may be captured by taking into account a number of selected variants that may be interacting with each other under an additive hypothesis